Archive for the ‘Nutrition’ Category

Tricked Into Getting Fat

Wednesday, May 27th, 2009

Fat plays by some pretty specific rules.

1. In humans, insulin is the most potent hormone inhibiting lipolysis (breakdown of fat for use as energy)

2. Another function of the hormone insulin is that it INCREASES lipogenesis (the synthesis of triglycerides from glucose/sugar). Triglycerides are the storage units of fats found in adipose tissue.

QUICK TRANSLATION=As long as we eat sugar and have insulin coursing through our bodies to deliver that sugar for energy use by cells, we will NOT burn fat…PERIOD! And really this means any sugar: fruit, breads, multi grains, pastas, ice cream, soda, chips, candy…they are all dealt with basically the same way.

But something is amiss here. Are we not told by the nutrition experts via media to eat a low fat, multi-grain, moderate protein diet for good health. Yeah… yeah we most certainly are.

But, we continue to play by the nutritional guidelines and rules… and at the same time losing the battle of the bulge and, we are losing miserably. Here’s why…  

A. We eat carbohydrates=any of a class of organic compounds composed of carbon, hydrogen, and oxygen, including starches and sugars  (Webster’s College Dictionary)

B. Our hypothalamus receives stimulating signals from a sudden increase in blood sugars and releases hormones that directs the organs of the body responsible for energy metabolism ( e.g. pancreas, liver, small intestine) to take this surplus of blood sugar and supply it for use  “right now” as an energy source for muscle contraction; stored in muscle for later use (glycogen); or stored as surplus energy source in the liver(also called glycogen).

It is important to note that only about 500 g (1.1 lb) of glycogen can be stored, 25% in the liver and 75% in muscle.

So, a few things to consider here: If your liver and muscles are replete with glycogen and you have sugar coursing through your circulation, there is a final resting place for the surplus…Adipose tissue or better know as FAT CELLS.

You see the regulating ”switch” for uptake of sugar into fat cells by insulin transport is almost always on. And, to make things even more interesting it is a VERY simple conversion process for adipose tissue to take  a sugar and store it as a triglyceride.

This puts the Kabosh on the whole grain, “good carb” low fat eating dogma the RDA has since the 1970’s and still to this day dispenses as the truth be told for a healthy lifestyle. 

I am going to go off tangent here, but this reminds me: Do you know how farmers get cattle big and fat and ready for slaughter so relatively quick and easy?

Grains!

What kind of grains?

 Corn! (Corn is a grain…not a vegetable) 

That’s right… Corn in feed lots…

Not sticks of fat layden butter or mounds of nuts in feed lots, but a low fat grain.

Cows have an extremely low fat diet.

So, how do they get so fat?

They have a high sugar diet that never gives their pancreas a rest because it has to constantly pump out insulin and drive all that sugar into their fat stores that course through and around their muscle. We eat that muscle as steak or burgers. That meat is considered by some nutritional purists as toxic…and really, it is. But, that is for another story…

To get back to the story here though that is exactly what we humans do everyday. We violate the rules of physiology and get tricked into a state of high grain, low fat eating and pay the price. Do you want to see a laundry list of paying the price for eating a carbohydrate based diet?  Alright, you asked for it

 

Contributed by Nancy Appleton, PhD
Author of the book Lick The Sugar Habit

In addition to throwing off the body’s homeostasis, excess sugar may result in a number of other significant consequences. The following is a listing of some of sugar’s metabolic consequences from a variety of medical journals and other scientific publications.

  1. Sugar can suppress your immune system and impair your defenses against infectious disease.1,2
  2. Sugar upsets the mineral relationships in your body: causes chromium and copper deficiencies and interferes with absorption of calcium and magnesium. 3,4,5,6
  3. Sugar can cause can cause a rapid rise of adrenaline, hyperactivity, anxiety, difficulty concentrating, and crankiness in children.7,8
  4. Sugar can produce a significant rise in total cholesterol, triglycerides and bad cholesterol and a decrease in good cholesterol.9,10,11,12
  5. Sugar causes a loss of tissue elasticity and function.13
  6. Sugar feeds cancer cells and has been connected with the development of cancer of the breast, ovaries, prostate, rectum, pancreas, biliary tract, lung, gallbladder and stomach.14,15,16,17,18,19,20
  7. Sugar can increase fasting levels of glucose and can cause reactive hypoglycemia.21,22
  8. Sugar can weaken eyesight.23
  9. Sugar can cause many problems with the gastrointestinal tract including: an acidic digestive tract, indigestion, malabsorption in patients with functional bowel disease, increased risk of Crohn’s disease, and ulcerative colitis.24,25,26,27,28
  10. Sugar can cause premature aging.29
  11. Sugar can lead to alcoholism.30
  12. Sugar can cause your saliva to become acidic, tooth decay, and periodontal disease.31,32,33
  13. Sugar contributes to obesity.34
  14. Sugar can cause autoimmune diseases such as: arthritis, asthma, multiple sclerosis.35,36,37
  15. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections).38
  16. Sugar can cause gallstones.39
  17. Sugar can cause appendicitis.40
  18. Sugar can cause hemorrhoids.41
  19. Sugar can cause varicose veins.42
  20. Sugar can elevate glucose and insulin responses in oral contraceptive users.43
  21. Sugar can contribute to osteoporosis.44
  22. Sugar can cause a decrease in your insulin sensitivity thereby causing an abnormally high insulin levels and eventually diabetes.45,46,47
  23. Sugar can lower your Vitamin E levels.48
  24. Sugar can increase your systolic blood pressure.49
  25. Sugar can cause drowsiness and decreased activity in children.50
  26. High sugar intake increases advanced glycation end products (AGEs)(Sugar molecules attaching to and thereby damaging proteins in the body).51
  27. Sugar can interfere with your absorption of protein.52
  28. Sugar causes food allergies.53
  29. Sugar can cause toxemia during pregnancy.54
  30. Sugar can contribute to eczema in children.55
  31. Sugar can cause atherosclerosis and cardiovascular disease.56,57
  32. Sugar can impair the structure of your DNA.58
  33. Sugar can change the structure of protein and cause a permanent alteration of the way the proteins act in your body.59,60
  34. Sugar can make your skin age by changing the structure of collagen.61
  35. Sugar can cause cataracts and nearsightedness.62,63
  36. Sugar can cause emphysema.64
  37. High sugar intake can impair the physiological homeostasis of many systems in your body.65
  38. Sugar lowers the ability of enzymes to function.66
  39. Sugar intake is higher in people with Parkinson’s disease.67
  40. Sugar can increase the size of your liver by making your liver cells divide and it can increase the amount of liver fat.68,69
  41. Sugar can increase kidney size and produce pathological changes in the kidney such as the formation of kidney stones.70,71
  42. Sugar can damage your pancreas.72
  43. Sugar can increase your body’s fluid retention.73
  44. Sugar is enemy #1 of your bowel movement.74
  45. Sugar can compromise the lining of your capillaries.75
  46. Sugar can make your tendons more brittle.76
  47. Sugar can cause headaches, including migraines.77
  48. Sugar can reduce the learning capacity, adversely affect school children’s grades and cause learning disorders.78,79
  49. Sugar can cause an increase in delta, alpha, and theta brain waves which can alter your mind’s ability to think clearly.80
  50. Sugar can cause depression.81
  51. Sugar can increase your risk of gout.82
  52. Sugar can increase your risk of Alzheimer’s disease.83
  53. Sugar can cause hormonal imbalances such as: increasing estrogen in men, exacerbating PMS, and decreasing growth hormone.84,85,86,87
  54. Sugar can lead to dizziness.88
  55. Diets high in sugar will increase free radicals and oxidative stress.89
  56. High sucrose diets of subjects with peripheral vascular disease significantly increases platelet adhesion.90
  57. High sugar consumption of pregnant adolescents can lead to substantial decrease in gestation duration and is associated with a twofold increased risk for delivering a small-for-gestational-age (SGA) infant.91,92
  58. Sugar is an addictive substance.93
  59. Sugar can be intoxicating, similar to alcohol.94
  60. Sugar given to premature babies can affect the amount of carbon dioxide they produce.95
  61. Decrease in sugar intake can increase emotional stability.96
  62. Your body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.97
  63. The rapid absorption of sugar promotes excessive food intake in obese subjects.98
  64. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).99
  65. Sugar adversely affects urinary electrolyte composition.100
  66. Sugar can slow down the ability of your adrenal glands to function.101
  67. Sugar has the potential of inducing abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases.102
  68. I.V.s (intravenous feedings) of sugar water can cut off oxygen to your brain.103
  69. Sugar increases your risk of polio.104
  70. High sugar intake can cause epileptic seizures.105
  71. Sugar causes high blood pressure in obese people.106
  72. In intensive care units: Limiting sugar saves lives.107
  73. Sugar may induce cell death.108
  74. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44 percent drop in antisocial behavior.109
  75. Sugar dehydrates newborns.110
  76. Sugar can cause gum disease.111

References

  1. Sanchez, A., et al. Role of Sugars in Human Neutrophilic Phagocytosis, American Journal of Clinical Nutrition. Nov 1973;261:1180_1184. Bernstein, J., al. Depression of Lymphocyte Transformation Following Oral Glucose Ingestion. American Journal of Clinical Nutrition.1997;30:613
  2. Ringsdorf, W., Cheraskin, E. and Ramsay R. Sucrose, Neutrophilic Phagocytosis and Resistance to Disease, Dental Survey. 1976;52(12):46_48.
  3. Couzy, F., et al. “Nutritional Implications of the Interaction Minerals,” Progressive Food and Nutrition Science 17;1933:65-87
  4. Kozlovsky, A., et al. Effects of Diets High in Simple Sugars on Urinary Chromium Losses. Metabolism. June 1986;35:515_518.
  5. Fields, M.., et al. Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets, Journal of Clinical Nutrition. 1983;113:1335_1345.
  6. Lemann, J. Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium. Journal of Clinical Nutrition. 1976 ;70:236_245.
  7. Goldman, J., et al. Behavioral Effects of Sucrose on Preschool Children. Journal of Abnormal Child Psychology.1986;14(4):565_577.
  8. Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7.
  9. Scanto, S. and Yudkin, J. The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and Body Weight in Human Volunteers, Postgraduate Medicine Journal. 1969;45:602_607.
  10. Albrink, M. and Ullrich I. H. Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High Carbohydrate Diets. American Journal of Clinical Nutrition. 1986;43:419-428. Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Med Hypotheses. Mar 1993;40(3):174-81.
  11. Reiser, S. Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease. Nutritional Health. 1985;203_216.
  12. Lewis, G. F. and Steiner, G. Acute Effects of Insulin in the Control of Vldl Production in Humans. Implications for The insulin-resistant State. Diabetes Care. 1996 Apr;19(4):390-3 R. Pamplona, M. .J., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses. 1990;40:174-181.
  13. Cerami, A., Vlassara, H., and Brownlee, M. “Glucose and Aging.” Scientific American. May 1987:90. Lee, A. T. and Cerami, A. The Role of Glycation in Aging. Annals of the New York Academy of Science; 663:63-67.
  14. Takahashi, E., Tohoku University School of Medicine, Wholistic Health Digest. October 1982:41:00
  15. Quillin, Patrick, Cancer’s Sweet Tooth, Nutrition Science News. Ap 2000 Rothkopf, M.. Nutrition. July/Aug 1990;6(4).
  16. Michaud, D. Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study. J Natl Cancer Inst. Sep 4, 2002 ;94(17):1293-300.
  17. Moerman, C. J., et al. Dietary Sugar Intake in the Etiology of Biliary Tract Cancer. International Journal of Epidemiology. Ap 1993.2(2):207-214.
  18. The Edell Health Letter. Sept 1991;7:1.
  19. De Stefani, E.”Dietary Sugar and Lung Cancer: a Case control Study in Uruguay.” Nutrition and Cancer. 1998;31(2):132_7.
  20. Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France. European Journal of Epidemiology 11 (1995):55-65.
  21. Kelsay, J., et al. Diets High in Glucose or Sucrose and Young Women. American Journal of Clinical Nutrition. 1974;27:926_936. Thomas, B. J., et al. Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to Oral Glucose, Human Nutrition Clinical Nutrition. 1983; 36C(1):49_51.
  22. Dufty, William. Sugar Blues. (New York:Warner Books, 1975).
  23. Acta Ophthalmologica Scandinavica. Mar 2002;48;25. Taub, H. Ed. Sugar Weakens Eyesight, VM NEWSLETTER;May 1986:06:00
  24. Dufty.
  25. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129
  26. Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France, European Journal of Epidemiology. 1995;11
  27. Persson P. G., Ahlbom, A., and Hellers, G. Epidemiology. 1992;3:47-52.
  28. Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7.
  29. Lee, A. T.and Cerami A. The Role of Glycation in Aging. Annals of the New York Academy of Science.1992;663:63-70.
  30. Abrahamson, E. and Peget, A. Body, Mind and Sugar. (New York: Avon, 1977.}
  31. Glinsmann, W., Irausquin, H., and Youngmee, K. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force. 1986:39:00 Makinen K.K.,et al. A Descriptive Report of the Effects of a 16_month Xylitol Chewing_gum Programme Subsequent to a 40_month Sucrose Gum Programme. Caries Research. 1998; 32(2)107_12.
  32. Glinsmann, W., Irausquin, H., and K. Youngmee. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force.1986;39:36_38.
  33. Appleton, N. New York: Healthy Bones. Avery Penguin Putnam:1989.
  34. Keen, H., et al. Nutrient Intake, Adiposity, and Diabetes. British Medical Journal. 1989; 1:00 655_658
  35. Darlington, L., Ramsey, N. W. and Mansfield, J. R. Placebo Controlled, Blind Study of Dietary Manipulation Therapy in Rheumatoid Arthritis, Lancet. Feb 1986;8475(1):236_238.
  36. Powers, L. Sensitivity: You React to What You Eat. Los Angeles Times. (Feb. 12, 1985). Cheng, J., et al. Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors. Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396.
  37. Erlander, S. The Cause and Cure of Multiple Sclerosis, The Disease to End Disease.” Mar 3, 1979;1(3):59_63.
  38. Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984).
  39. Heaton, K. The Sweet Road to Gallstones. British Medical Journal. Apr 14, 1984; 288:00:00 1103_1104. Misciagna, G., et al. American Journal of Clinical Nutrition. 1999;69:120-126.
  40. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).
  41. Ibid.
  42. Cleave, T. and Campbell, G. (Bristol, England:Diabetes, Coronary Thrombosis and the Saccharine Disease: John Wright and Sons, 1960).
  43. Behall, K. Influ ence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters. Disease Abstracts International. 1982;431437.
  44. Tjäderhane, L. and Larmas, M. A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats. Journal of Nutrition. 1998:128:1807_1810.
  45. Beck, Nielsen H., Pedersen O., and Schwartz S. Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects. Diabetes. 1978;15:289_296 .
  46. Sucrose Induces Diabetes in Cat. Federal Protocol. 1974;6(97). diabetes
  47. Reiser, S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans. American Journal of Clinical Nutrition. 1986;43:151-159.
  48. Journal of Clinical Endocrinology and Metabolism. Aug 2000
  49. Hodges, R., and Rebello, T. Carbohydrates and Blood Pressure. Annals of Internal Medicine. 1983:98:838_841.
  50. Behar, D., et al. Sugar Challenge Testing with Children Considered Behaviorally Sugar Reactive. Nutritional Behavior. 1984;1:277_288.
  51. Furth, A. and Harding, J. Why Sugar Is Bad For You. New Scientist. Sep 23, 1989;44.
  52. Simmons, J. Is The Sand of Time Sugar? LONGEVITY. June 1990:00:00 49_53.
  53. Appleton, N. New York: LICK THE SUGAR HABIT. Avery Penguin Putnam:1988. allergies
  54. Cleave, T. The Saccharine Disease: (New Canaan Ct: Keats Publishing, Inc., 1974).131.
  55. Ibid. 132
  56. Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses . 1990:00:00 174_181.
  57. Vaccaro O., Ruth, K. J. and Stamler J. Relationship of Postload Plasma Glucose to Mortality with 19 yr Follow up. Diabetes Care. Oct 15,1992;10:328_334. Tominaga, M., et al, Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose. Diabetes Care. 1999:2(6):920-924.
  58. Lee, A. T. and Cerami, A. Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging. Handbook of the Biology of Aging. (New York: Academic Press, 1990.).
  59. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology 1990:45(4):105_110.
  60. Cerami, A., Vlassara, H., and Brownlee, M. Glucose and Aging. Scientific American. May 1987:00:00 90
  61. Dyer, D. G., et al. Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging. Journal of Clinical Investigation. 1993:93(6):421_22.
  62. Veromann, S.et al.”Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development.” Ophthalmologica. 2003 Jul-Aug;217(4):302-307.
  63. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI
  64. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4):105_110.
  65. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
  66. Appleton, Nancy. New York; Lick the Sugar Habit. Avery Penguin Putnam, 1988 enzymes
  67. Hellenbrand, W. Diet and Parkinson’s Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study. Neurology. Sep 1996;47(3):644-650.
  68. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38.
  69. Ibid.
  70. Yudkin, J., Kang, S. and Bruckdorfer, K. Effects of High Dietary Sugar. British Journal of Medicine. Nov 22, 1980;1396.
  71. Blacklock, N. J., Sucrose and Idiopathic Renal Stone. Nutrition and Health. 1987;5(1-2):9- Curhan, G., et al. Beverage Use and Risk for Kidney Stones in Women. Annals of Internal Medicine. 1998:28:534-340.
  72. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI,:
  73. Ibid. fluid retention
  74. Ibid. bowel movement
  75. Ibid. compromise the lining of the capillaries
  76. Nash, J. Health Contenders. Essence. Jan 1992; 23:00 79_81.
  77. Grand, E. Food Allergies and Migraine.Lancet. 1979:1:955_959.
  78. Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981.)
  79. Molteni, R, et al. A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning. NeuroScience. 2002;112(4):803-814.
  80. Christensen, L. The Role of Caffeine and Sugar in Depression. Nutrition Report. Mar 1991;9(3):17-24.
  81. Ibid,44
  82. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129
  83. Frey, J. Is There Sugar in the Alzheimer’s Disease? Annales De Biologie Clinique. 2001; 59 (3):253-257.
  84. Yudkin, J. Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes. Nutrition and Health. 1987;5(1-2):5-8.
  85. Yudkin, J and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988:32(2):53-55.
  86. The Edell Health Letter. Sept 1991;7:1.
  87. Gardner, L. and Reiser, S. Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and Cortisol. Proceedings of the Society for Experimental Biology and Medicine. 1982;169:36_40.
  88. Journal of Advanced Medicine. 1994;7(1):51-58.
  89. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
  90. Postgraduate Medicine.Sept 1969:45:602-07.
  91. Lenders, C. M. Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant Adolescents. Journal of Nutrition. Jun 1997;1113- 1117
  92. Ibid.
  93. Sugar, White Flour Withdrawal Produces Chemical Response. The Addiction Letter. Jul 1992:04:00 Colantuoni, C., et al. Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence. Obes Res. Jun 2002 ;10(6):478-488. Annual Meeting of the American Psychological Society, Toronto, June 17, 2001 http://articles.mercola.com/sites/articles/archive/2001/06/30/sugar-part-two.aspx
  94. Ibid.
  95. Sunehag, A. L., et al. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition Diabetes. 1999 ;48 7991_800.
  96. Christensen L., et al. Impact of A Dietary Change on Emotional Distress. Journal of Abnormal Psychology.1985;94(4):565_79.
  97. Nutrition Health Review. Fall 85 changes sugar into fat faster than fat
  98. Ludwig, D. S., et al. High Glycemic Index Foods, Overeating and Obesity. Pediatrics. March 1999;103(3):26-32.
  99. Pediatrics Research. 1995;38(4):539-542. Berdonces, J. L. Attention Deficit and Infantile Hyperactivity. Rev Enferm. Jan 2001;4(1)11-4
  100. Blacklock, N. J. Sucrose and Idiopathic Renal Stone. Nutrition Health. 1987;5(1 & 2):9-
  101. Lechin, F., et al. Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans. Neurophychobiology. 1992;26(1-2):4-11.
  102. Fields, M. Journal of the American College of Nutrition. Aug 1998;17(4):317_321.
  103. Arieff, A. I. Veterans Administration Medical Center in San Francisco. San Jose Mercury; June 12/86. IVs of sugar water can cut off oxygen to the brain.
  104. Sandler, Benjamin P. Diet Prevents Polio. Milwakuee, WI,:The Lee Foundation for for Nutritional Research, 1951
  105. Murphy, Patricia. The Role of Sugar in Epileptic Seizures. Townsend Letter for Doctors and Patients. May, 2001 Murphy Is Editor of Epilepsy Wellness Newsletter, 1462 West 5th Ave., Eugene, Oregon 97402
  106. Stern, N. & Tuck, M. Pathogenesis of Hypertension in Diabetes Mellitus. Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (PhiladelphiA; A:Lippincott Williams & Wilkins, 2000)943-957.
  107. Christansen, D. Critical Care: Sugar Limit Saves Lives. Science News. June 30, 2001; 159:404.
  108. Donnini, D. et al. Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.Biochem Biohhys Res Commun. Feb 15, 1996:219(2):412-417.
  109. Schoenthaler, S. The Los Angeles Probation Department Diet-Behavior Program: Am Empirical Analysis of Six Institutional Settings. Int J Biosocial Res 5(2):88-89.
  110. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes. 1999 Apr;48(4):791-800.
  111. Glinsmann, W., et al. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners.” FDA Report of Sugars Task Force -1986 39 123 Yudkin, J. and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988;32(2):53-5.

Now for the $64,000 question.

How do we get rid of fat and get strong for LIFE?

Easy. There are two ”choices” we can make right now to get on the fast track to a fat free and strong life- 

1. Eat lean, grass fed meat when you can, free range chicken and eggs, wild caught fish, local grown and/or organic fruits and vegetables, raw nuts and seeds, water.  

This sounds ridiculously impractical and unrealistic I know. But, really these are the foods our bodies thrive and depend on for optimal health. This is not something we should waste too much time in refuting, though. It would be almost absurd if we were to contest these particular foods as not being the answer to all of our health concerns. But we are 21st century humans who want it all without leaving our certain comfort zones. And we will find it practical to disregard the truth so blatant and exposed for us and rather replace it with any of the common excuses so readily available to convenience our decadent pleasures.  That is just the way it goes, I guess.  And with that said, here is  the other choice-

2. Exercise. And not an aerobics class exercise program. The type of exercise program that your body needs, craves and will absolutely shed the fat and get you fit like no other is a program that will challenge you every time. A program like  WWW.MATTJENNINGSBOOTCAMPS.COM

Why is this an exercise program you should consider? Because WWW.MATTJENNINGSBOOTCAMPS.COM is an exercise program designed to amp up your metabolism for extreme fat loss and lean muscle building by stimulating hormones that cause  high level lipolysis (fat breakdown for use as fuel/energy), and at the same time preserves lean muscle for a beautifully sculpted body.

Hey, these are choices. And if the prior choices you have made are not getting you the results you deserve then the information provided in this post maybe the answers that have eluded you for far too long. No more excuses because you are now in the know.  And knowing and doing are two different things. But hey, you already know that.

Peace-

Matt

 

    

    

Good Health By Choice

Sunday, December 28th, 2008

Before I ever heard of Atkins, Sears, Eades, Cordain… I had a very smart nutritional “guru” fill me in on

the “right way to eat”. His name was dad. And he gave me and my brothers and sister the greatest gift

he could have ever given us…the advice  to not eat sugar.

I was probably 10 years old (33 years ago) when he began his well intentioned enlightenment for me about exercise and nutrition. And to more accurately describe his abhorrent depiction of sugar I believe his exact words when on the topic were “SUGAR IS POISON.” 

Yeah, he made no bones about it. And I was fortunate to take this advice for the long haul and do my best to share it as tactfully as I could bare with whomever wanted to listen.

Well, today I can share that more than ever, his wisdom proves true with me as well as through science. For me, an empirical reminder that when you do not obey the rules of proper nourishment, you will pay the price.

Right before the Christmas holidays, I endulged in some homemade cookies that seem to be so ubiquitous this time of year. That turned into a little pasta here and a slice of homemade breads there and walla, kick ass sinus infection that has found me dizzy with vertigo type symptoms for over a week. And you know what, I knew that if I chose to eat these types of “foods” I should be willing to pay the price. If feeling like the top half of my head is about to explode and that I am on an all day “TILT-A-WHIRL” ride at the amusement park whos operator is laughing his ass off and won’t stop the ride is paying the price, then I am flat out broke paying the nutrition reaper and I want off this ride NOW!

Yesterday I went back on my normal HUNTER-GATHERER approach to eating and the ride is slowing down for sure and my head is getting clearer. That is why I am now writing this.

The season to get on track with nutrition and exercise doesn’t start AFTER the holidays. It starts RIGHT NOW! Get back on track with me and get sickness out of your life.

But hey, if you are willing to pay the price thats cool. Just read what might be in store for you if you continue on the “sugar train”:

76 Ways Sugar Can Ruin Your Health (WWW.MERCOLA.COM)

 

Contributed by Nancy Appleton, Ph.D
Author of the book Lick The Sugar Habit

In addition to throwing off the body’s homeostasis, excess sugar may result in a number of other significant consequences. The following is a listing of some of sugar’s metabolic consequences from a variety of medical journals and other scientific publications.

  1. Sugar can suppress your immune system and impair your defenses against infectious disease.1,2
  2. Sugar upsets the mineral relationships in your body: causes chromium and copper deficiencies and interferes with absorption of calcium and magnesium. 3,4,5,6
  3. Sugar can cause can cause a rapid rise of adrenaline, hyperactivity, anxiety, difficulty concentrating, and crankiness in children.7,8
  4. Sugar can produce a significant rise in total cholesterol, triglycerides and bad cholesterol and a decrease in good cholesterol.9,10,11,12
  5. Sugar causes a loss of tissue elasticity and function.13
  6. Sugar feeds cancer cells and has been connected with the development of cancer of the breast, ovaries, prostate, rectum, pancreas, biliary tract, lung, gallbladder and stomach.14,15,16,17,18,19,20
  7. Sugar can increase fasting levels of glucose and can cause reactive hypoglycemia.21,22
  8. Sugar can weaken eyesight.23
  9. Sugar can cause many problems with the gastrointestinal tract including: an acidic digestive tract, indigestion, malabsorption in patients with functional bowel disease, increased risk of Crohn’s disease, and ulcerative colitis.24,25,26,27,28
  10. Sugar can cause premature aging.29
  11. Sugar can lead to alcoholism.30
  12. Sugar can cause your saliva to become acidic, tooth decay, and periodontal disease.31,32,33
  13. Sugar contributes to obesity.34
  14. Sugar can cause autoimmune diseases such as: arthritis, asthma, multiple sclerosis.35,36,37
  15. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections)38
  16. Sugar can cause gallstones.39
  17. Sugar can cause appendicitis.40
  18. Sugar can cause hemorrhoids.41
  19. Sugar can cause varicose veins.42
  20. Sugar can elevate glucose and insulin responses in oral contraceptive users.43
  21. Sugar can contribute to osteoporosis.44
  22. Sugar can cause a decrease in your insulin sensitivity thereby causing an abnormally high insulin levels and eventually diabetes.45,46,47
  23. Sugar can lower your Vitamin E levels.48
  24. Sugar can increase your systolic blood pressure.49
  25. Sugar can cause drowsiness and decreased activity in children.50
  26. High sugar intake increases advanced glycation end products (AGEs)(Sugar molecules attaching to and thereby damaging proteins in the body).51
  27. Sugar can interfere with your absorption of protein.52
  28. Sugar causes food allergies.53
  29. Sugar can cause toxemia during pregnancy.54
  30. Sugar can contribute to eczema in children.55
  31. Sugar can cause atherosclerosis and cardiovascular disease.56,57
  32. Sugar can impair the structure of your DNA.58
  33. Sugar can change the structure of protein and cause a permanent alteration of the way the proteins act in your body.59,60
  34. Sugar can make your skin age by changing the structure of collagen.61
  35. Sugar can cause cataracts and nearsightedness.62,63
  36. Sugar can cause emphysema.64
  37. High sugar intake can impair the physiological homeostasis of many systems in your body.65
  38. Sugar lowers the ability of enzymes to function.66
  39. Sugar intake is higher in people with Parkinson’s disease.67
  40. Sugar can increase the size of your liver by making your liver cells divide and it can increase the amount of liver fat.68,69
  41. Sugar can increase kidney size and produce pathological changes in the kidney such as the formation of kidney stones.70,71
  42. Sugar can damage your pancreas.72
  43. Sugar can increase your body’s fluid retention.73
  44. Sugar is enemy #1 of your bowel movement.74
  45. Sugar can compromise the lining of your capillaries.75
  46. Sugar can make your tendons more brittle.76
  47. Sugar can cause headaches, including migraines.77
  48. Sugar can reduce the learning capacity, adversely affect school children’s grades and cause learning disorders.78,79
  49. Sugar can cause an increase in delta, alpha, and theta brain waves which can alter your mind’s ability to think clearly.80
  50. Sugar can cause depression.81
  51. Sugar can increase your risk of gout.82
  52. Sugar can increase your risk of Alzheimer’s disease.83
  53. Sugar can cause hormonal imbalances such as: increasing estrogen in men, exacerbating PMS, and decreasing growth hormone.84,85,86,87
  54. Sugar can lead to dizziness.88
  55. Diets high in sugar will increase free radicals and oxidative stress.89
  56. High sucrose diets of subjects with peripheral vascular disease significantly increases platelet adhesion.90
  57. High sugar consumption of pregnant adolescents can lead to substantial decrease in gestation duration and is associated with a twofold increased risk for delivering a small-for-gestational-age (SGA) infant.91,92
  58. Sugar is an addictive substance.93
  59. Sugar can be intoxicating, similar to alcohol.94
  60. Sugar given to premature babies can affect the amount of carbon dioxide they produce.95
  61. Decrease in sugar intake can increase emotional stability.96
  62. Your body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.97
  63. The rapid absorption of sugar promotes excessive food intake in obese subjects.98
  64. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).99
  65. Sugar adversely affects urinary electrolyte composition.100
  66. Sugar can slow down the ability of your adrenal glands to function.101
  67. Sugar has the potential of inducing abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases.102
  68. I.V.s (intravenous feedings) of sugar water can cut off oxygen to your brain.103
  69. Sugar increases your risk of polio.104
  70. High sugar intake can cause epileptic seizures.105
  71. Sugar causes high blood pressure in obese people.106
  72. In intensive care units: Limiting sugar saves lives.107
  73. Sugar may induce cell death.108
  74. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44 percent drop in antisocial behavior.109
  75. Sugar dehydrates newborns.110
  76. Sugar can cause gum disease.111

Why Not Grains?

Saturday, December 13th, 2008

I found and linked a very interesting site that supports hunter-gatherer philosophies in nutrition (the same ones that I practice). So if you need even more evidence about the harms of grains in the human diet, PLEASE read this link http://www.beyondveg.com/cordain-l/grains-leg/grains-legumes-1a.shtml

Have an awesome weekend…

and if you want to discuss…that would be cool

Peace-Matt

 

 

Why is Matt Doing This?

Friday, October 10th, 2008

Because, ALL of you deserve “killer” info.  Hopefully everyone checked out last night’s e-mail.

Now, for “my boy” Dr. Scott Lloyd to tag up and send the message. Dr. Scott knows his stuff and any kick butt information he sends my way is going out there for you to devour.

                So, here we go Dr. Scott… It’s time to…

                              ripp it up

Peace-

Matt 

 

 

 

Most People, especially physicians, are unaware of what we require genetically on a cellular level for optimal health. Genetically we are programmed to eat two food groups - 1) fruit and vegetables and 2) Meat and Fish

reference: Eaton, S Boyd M.D. & Konner, M PhD. Paleolithic Nutrition: A consideration of its nature and current implications. 1985: New England Journal of Medicine 312, 283-289 (researchers warned us of the dangers of low fat high carbohydrate back in 1985!!!)

 

Also, as you know, commercial meat and true wild meat are very different in chemical composition. The studies that ‘prove’ that meat is no good for us are always referencing commercial meat (full of omega 6, growth hormones, antibiotics, bioaccumulation of herbicides/pesticides/and other drugs, etc) rather than wild meat or organic grass fed free range meat.

reference: James Chestnut MSc., D.C. “_The Innate Diet and Natural Hygiene_” copyright 2004 The Wellness Practice - Global Self Health Corporation.

 

Anyway, I wanted to help reinforce to people the importance of what you are sharing with them. Think about this for a minute: What do they feed animals to fatten them up? Answer: Grain. America has become a feed lot and we are paying the price.

 

Lastly, for those with cholesterol concerns go to:

http://www.ravnskov.nu/cholesterol.htm

http://www.thincs.org/

http://easydiagnosis.com/controversies.html

http://www.second-opinions.co.uk/

http://www.avoidheartattack.com/

 

Thanks again Matt for helping us learn and grow, so we may be at our best

 

Many blessings

Scott

 

Scott Lloyd, Chiropractor

Cafe of Life

Helping Families Create Health, Happiness, and Well Being!

www.scottlloyd.info

 

 

Food For Thought

Friday, October 10th, 2008

I just want to share with you the concepts in nutrition that I have been following for about 14 years now. It has served me well and it has served everyone that I have introduced to the low-carb lifestyle very well, also. The idea is simple in that eating carbohydrates, and these are the usual suspects… grains, breads, pasta, potatoes, large amounts of fruit, simple sugars, complex sugars…puts your metabolism in “storage” mode and basically gets you fat as well as just wreaks havoc on your body causing plethora of dysfunction in ways that you would not even think i.e. arthritis, asthma, CVD, depression, high triglycerides…the list is really expansive.  

But, instead of me explaining some of the research that backs up VLCKD- I’ll leave that to “the man” Dr. Jeff Volek. So, please read and enjoy these 2 articles

 

Tomorrow is another gift to ripp it up

 Peace-

Matt matt@shiftxtr.com

 

P.S. It is curious that the people who disparage these aforementioned  concepts are the same people who have high blood pressure, high triglycerides, are usually overweight etc., and are on a number of toxic meds to “treat” these problems. And their doctors are seemingly fine with keeping this insidious cycle rolling. Scary stuff, man. Scary, scary stuff.

 

Shocking Study: Eating Saturated Fat Healthier For You Than Restricting It

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by Jimmy Moore   

Saturday, 29 December 2007


Dr. Jeff Volek says controlling insulin is vital to blood lipidsA little over two years ago, I shared with you this study on the connection between metabolic syndrome and livin’ la vida low-carb. The researchers were Dr. Jeff Volek from the University of Connecticut and Dr. Richard Feinman from SUNY Downstate Medical Center in Brooklyn, NY and they were absolutely fascinated by the rather obvious intertwining of a natural dietary approach like low-carb improving virtually every single area of metabolic syndrome (a precursor to diabetes, heart disease and stroke), including obesity, high triglycerides, low HDL “good” cholesterol, high blood sugar, hypertension and insulin resistance.Now there is brand new research from these same two researchers with something quite startling regarding a comparison between a low-fat and a low-carb diet as it relates to inflammation and saturated fat in the bloodstream. 

 

Lead researcher Dr. Jeff S. Volek, PhD, RD from the Department of Kinesiology at the University of Connecticut and his team of outstanding researchers (including Dr. Feinman, Dr. Stephen Phinney, and the soon-to-be Dr. Cassandra Forsythe, among others) tested the various components of metabolic syndrome comparing a carbohydrate-restricted diet with a low-fat diet in overweight men and women over a 12-week period. The study participants were split into one of two groups:

 

 

VLCKD (very low-carb ketogenic diet)–1504 calories
Fat/Protein/Carbohydrate ratio of 59/28/12

 

OR

LFD (low-fat diet)–1478 calories
Fat/Protein/Carbohydrate ration of 24/20/56

What did Dr. Volek and his team of researchers find?

Total saturated fatty acids in the blood actually DECREASED in the VLCKD group while the anti-inflammatory markers also “significantly decreased.” Meanwhile, the LFD group, which consumed two-thirds less saturated fat than the VLCKD group, saw an INCREASE in total saturated fat in the bloodstream despite reducing fat intake.

This was totally unexpected as the conventional wisdom regarding saturated fat consumption is that it causes an increase in inflammation which leads to a worsening of the metabolic syndrome conditions and overall health. But that’s not what happened.

“A very low carbohydrate diet resulted in profound alterations in fatty acid composition and reduced inflammation compared to a low fat diet,” the researchers concluded.

This study was published in the November 29, 2007 issue of the scientific journal Lipids.

So what are we to make of this research in light of all we’ve ever been told about saturated fat? Doctors and nutritionists have long told their patients with metabolic syndrome symptoms to eat a low-fat diet and now science like this one is showing the shortsightedness of this unproven recommendation. Livin’ la vida low-carb is making great strides behind-the-scenes because it is an excellent way to reduce triglycerides and other essential health markers related to inflammation.

Dr. Volek says this new study shows how a controlled-carbohydrate nutritional approach is “adding to the evolving picture of improvement in general health beyond simple weight loss in keeping blood glucose and insulin under control.” And he believes this hyperinsulinemia is the root cause behind obesity, diabetes, and a whole host of other preventable diseases that all improve with the use of a low-carb diet.

Interestingly, the Volek study in Lipids is only a small portion of a much larger study currently under peer review. The full study shows even more improvements in blood lipids (cholesterol) with the stunning conclusion that “lowering total and saturated fat only had a small effect on circulating inflammatory markers whereas reducing carbohydrate led to considerably greater reductions in a number of pro-inflammatory” markers. Dr. Volek says this puts the onus of health risks back on the consumption of carbohydrates.

“These data implicate dietary carbohydrate rather than fat as a more significant nutritional factor contributing to inflammatory processes,” he stated.

Meanwhile, Richard Feinman, PhD from the biochemistry department at SUNY Downstate Medical Center says this new research demonstrably shows why carb-restricted diets work so remarkably well.

“The real importance of diets that lower carbohydrate content is that they are grounded in mechanism: carbohydrates stimulate insulin secretion which biases fat metabolism towards storage rather than oxidation,” Dr. Feinman explained. “The inflammation results open a new aspect of the problem. From a practical standpoint, continued demonstrations that carbohydrate restriction is more beneficial than low fat could be good news to those wishing to forestall or manage the diseases associated with metabolic syndrome.”

Most damning against the low-fat diet hypothesis is the fact that although there was a three-fold higher saturated fat consumption by the VLCKD group, it was the LFD group that experienced higher saturated fat in the blood. Counterintuitive? You betcha!

“This clearly shows the limitations of the idea that ‘you are what you eat,’” Dr. Volek explained. “Metabolism plays a big role. You are what your body does with what you eat.”

I like that–YOU ARE WHAT YOUR BODY DOES WITH WHAT YOU EAT! And that’s why I’m livin’ la vida low-carb because I have all the confidence in the world with what my body will do with the low-carb foods I consume. Controlled blood sugar and insulin levels, reduced triglycerides, lower blood pressure, increased HDL “good” cholesterol, and so much more than I could have ever expected from a high-fat, low-carb diet. It’s hard not to appreciate something like this when your life has been so radically changed for the better. Now the research is showing us why.

Dr. Feinman succinctly repeated and summarized what I blogged about in this previous post regarding saturated fat consumption on the low-carb lifestyle in the following statement about this new study.

“I think even if you allow for tremendous error, it says that if carbs are low, saturated fat doesn’t have much effect on the plasma composition,” he remarked.

And that is why I don’t worry about how much saturated fat I consume as long as my carbs are reduced. Now we have the science to back us up!

You can share your appreciation to Dr. Jeff Volek for his fantastic research by e-mailing him at jeff.volek@uconn.edu This e-mail address is being protected from spam bots, you need JavaScript enabled to view it as well as Dr. Richard Feinman at rfeinman@downstate.edu This e-mail address is being protected from spam bots, you need JavaScript enabled to view it .

 

 

Science News

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Low-carb Diet Reduces Inflammation And Blood Saturated Fat In Metabolic Syndrome

ScienceDaily (Dec. 4, 2007) — Metabolic syndrome is a condition afflicting one quarter to one third of adult men and women and is an established pre-cursor to diabetes, coronary heart disease, and other serious illnesses. Patients have long been advised to eat a low-fat diet even though carbohydrate restriction has been found to be more effective at reducing specific markers, such as high triglycerides, characteristic of the syndrome. Now, a new study indicates that a diet low in carbohydrates is also more effective than a diet low in fat in reducing saturated fatty acids in the blood and reducing markers of inflammation.


See also:

Health & Medicine

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Reference

Right-click here to download pictures. To help protect your privacy, Outlook prevented automatic download of this picture from the Internet. *                     Saturated fat

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Right-click here to download pictures. To help protect your privacy, Outlook prevented automatic download of this picture from the Internet. *                     Diabetic diet

While there have been contradictory and confusing messages directed at health conscious consumers about dietary recommendations, most researchers agree on the need to limit inflammatory agents. In a report published in the on-line version of the journal Lipids, researchers at the University of Connecticut with co-authors from SUNY Downstate Medical Center in Brooklyn, the University of Minnesota, and the University of California show much greater improvement in inflammatory markers in patients with metabolic syndrome on a very low carbohydrate approach compared to a low fat diet.

Lead researcher Jeff S. Volek, PhD, RD, associate professor of kinesiology at the University of Connecticut, describes the study as “adding to the evolving picture of improvement in general health beyond simple weight loss in keeping blood glucose and insulin under control.” The work is part of a larger study (currently under review) showing numerous improvements in blood lipids. The current work concludes that “lowering total and saturated fat only had a small effect on circulating inflammatory markers whereas reducing carbohydrate led to considerably greater reductions in a number of pro-inflammatory cytokines, chemokines, and adhesion molecules. These data implicate dietary carbohydrate rather than fat as a more significant nutritional factor contributing to inflammatory processes.”

Richard Feinman, PhD, professor of biochemistry at SUNY Downstate Medical Center, adds, “The real importance of diets that lower carbohydrate content is that they are grounded in mechanism — carbohydrates stimulate insulin secretion which biases fat metabolism towards storage rather than oxidation. The inflammation results open a new aspect of the problem. From a practical standpoint, continued demonstrations that carbohydrate restriction is more beneficial than low fat could be good news to those wishing to forestall or manage the diseases associated with metabolic syndrome.”

One of the remarkable effects in the data presented that may have contributed to the results is that despite the three-fold greater saturated fat in the diet for the low carb group, saturated fat in the blood turned out to be higher in the low fat group due to the process known as carbohydrate-induced lipogenesis. Dr. Volek points out that “this clearly shows the limitations of the idea that ‘you are what you eat.’ Metabolism plays a big role. You are what your body does with what you eat.”

.


Adapted from materials provided by Suny Downstate Medical Center, via EurekAlert!, a service of AAAS.

 

APA


MLA

Suny Downstate Medical Center (2007, December 4). Low-carb Diet Reduces Inflammation And Blood Saturated Fat In Metabolic Syndrome. ScienceDaily. Retrieved October 9, 2008, from http://www.sciencedaily.com­ /releases/2007/12/071203091236.htm

 

 

This one gets me everytime!

Sunday, September 21st, 2008

Good morning everyone in the fit world-

I have to just crush this myth on fat loss. I know you guys are sick of me bringing this topic up, but I am such a big proponent of getting and staying lean and healthy for myriad of reasons. And they are mostly health related. From heart health, metabolic health, lowering your risk or maybe even ELIMINATING risk for cancer, to strengthening your immune system and staying in a mental balance has everything to do with one major thing…GET SUGAR OUT OF YOUR DIET! There is no compromise here. This is about getting healthy and staying healthy for the long haul. I must be getting on your nerves about this but you know what? I don’t care. It is Sunday morning and I have fun plans with my family today. But, if I did not care about every one of you, I would simply not waste my time writing this e-mail.

I saw on the news earlier this morning an interview with the Editor of PREVENTION MAGAZINE. One of the first things she said during her “health” segment just ticked me off to the point I had to “correct” her just in case anyone of you saw the same segment.

She said- “Fat burns in the flame of carbohydrates.” Now, before I understood thermodynamics in metabolism and the influence of certain hormones on the storage and breakdown of “fuels” for energy, I thought- “what the hell does fat burns in the flame of carbohydrates (sugars) mean?” This just NEVER made sense to me. And, when I studied physiology in school and had discussions with some of the heavy duty researchers (Dr.Jeff Volek, Dr. James Chestnut, Dr. Joseph Mercola, Dr. Marsha Bradley to name a few) on this subject, it became clear that it didn’t make sense because it was a crock to begin with.

Let me simply state an absolute fact here and certainly anyone can argue this till they are blue in the face. But, “straight up”-

As long as metabolism is under direct influence of insulin and hence carbohydrate intake, YOU WILL NOT and YOU CAN NOT BURN FAT! This is just the way it goes.

Maybe asking this question will make even more sense- How can a body use fat as a fuel if that body is in storage mode. Insulin puts a body in storage mode. It really is that simple.  

Understand the influence of food. Get into a kick butt exercise program like the SHIFT BOOT CAMPS or SHIFT XTR training systems and you are on your way to a “kick butt life.”

Shouldn’t be any other life, now should it?

If anyone has any questions or anything they want to discuss with me, I am here 24/7 to hear you out. Even if you disagree with me, that’s cool. Let’s talk about it.

And, don’t forget about THE PREMIER HEALTH AND FITNESS MAGAZINE that is soon going to propel NEW JERSEYS health, fitness and wellness scene to NEW HEIGHTS…

“FITNESS FOCUS NEW JERSEY” Magazine will be the source we have all been waiting for!

 

Peace-

Matt matt@shiftxtr.com

 

     

No Dairy?

Saturday, September 13th, 2008

I thought that this might interest some of you. It is a response to a question from one of my boot camp members.

She was curious as to why my 30 Day Nutrition Plan did not contain dairy. This response is a somewhat edited version.

Please read-

 

I do not mention dairy simply because it is non existent in my eating. And the best reason I have is that I learned years ago that the only milk that we are ever to consume is from our mother’s breast. And once our mother stops lactating, that should be the end of milk consumption as we know it. The understanding is that a mother’s milk has all the nutrients and antibodies that initially support our young immune systems, specifically in our guts. This is also our first and most important exposure to antigens that further stimulates production of antibodies that helps us fight off disease. That is it. But, I can’t stop here without expanding the idea of why we should not consume any dairy products.

Humans are the only species in the animal kingdom that considers the consumption of another species milk. This is a very important observation that most have simply never considered.

Dairy is pasteurized, and during this heating process all of the enzymes that would help us digest the lactose (sugar) in milk and absorb any significant amount of calcium from dairy is destroyed. That main enzyme is called phosphataze.

Have you ever noticed how mucous tends to build up in our saliva after consuming dairy? We simply do not have the enzyme in our bodies to breakdown the proteins and sugar in dairy. That is very telling in my eyes.

When we consume dairy the mucosal lining of our gut thickens and this thickening disrupts proper absorption of many nutrients from many other foods.

There have been large studies on osteoporosis and calcium from dairy and calcium supplements and the results are basically that both are poorly absorbed and do very little to prevent osteoporosis.

You have to consider the huge lobbying for dairy farmers in the U.S. and the fact that the calcium supplement business is a multi-billion dollar industry.

So, considering what you have just read, and there is much more info. out there than just this, I would not consider dairy as a nutritional or nourishing food group. Ever!

 

This next point goes a little off tangent, but still a relevant idea. Consider this for a second; if you spent a significant amount of time in zero gravity, what would happen to your bone density? You would have significant mineral loss from bone and hence an osteoporosis type of state, although temporary.( And this response in bone loss would be regardless of any dietary calcium you may be consuming in zero gravity.) But as soon as you apply load to that bone after leaving zero gravity you stimulate bone growth. And, load bearing is the most important component in retaining bone health. As far as supporting mineral (calcium +)   absorption from the foods we eat, the best foods are dark green, leafy vegetables and legumes. Greens and beans. Greens would be collard, kale, asparagus, brussel sprouts…dark leaf cabbage. Even carrots contain calcium. The list is fairly long, but the absorption of calcium and other important minerals from vegetables is sufficient.

EXERCISE AND DARK GREEN VEGETABLES. This is the best way to remain bone healthy and strong.

 

Peace-

Matt matt@shiftxtr.com

Executive Editor

Fitness Focus New Jersey Magazine

        www.fitnessfocusnj.com

Weight Loss/Fat Loss-Not The Same

Thursday, July 24th, 2008

I want to quickly cover a misunderstanding in fat loss. Notice that I did not say weight loss. There is disparity between the two concepts that can get a little confusing so, in the interest of keeping tonight’s topic hopefully short and sweet, I will simply state a few physiological rules that will help guide you to the promised land of FAT LOSS once and for all.  The key here is to understand that FACTS can not be shoved aside. As an old buddy of mine likes to say-“It is what it is”, so here we go.

 

In both rats and humans, insulin is the most potent hormone inhibiting lipolysis. (Translation: Eating anything carbs.except most vegetables slams on the brakes for fat loss) Exercise Metabolism (Human Kinetics) Turcotte, Richter, and Kiens-Lipid Metabolism in Exercise.

The essential hormonal changes promoting increased lipolysis (the release and breakdown of fats from adipose tissue to be used as fuel) during whole-body exercise are increased sympathoadrenal B-adrenergic stimulation (adrenalin) and decreased circulating insulin levels.  This may sound a little complicated but an example that would demonstrate this response would be if you sprinted 100 meters as fast as you could. Heavy breathing, heart just pounding, legs burning …

Think OLYMPIC sprinter-ripped, muscular, fast.

 

And this study from the Annals of Internal Medicine

A LOW-CARBOHYDRATE, KETOGENIC DIET vs. A LOW-FAT DIET TO TREAT OBESITY AND HYPERLIPIDEMIA

William S. Yancy Jr., MD, MHS; Maren K. Olsen, PhD; John R Guyton, MD; Ronna P. Bakst, RD; and Eric C. Westman, MD MHS

 

A calorie is a calorie- The assumption would be that with the same meal frequency, as long as we adjust the calories in vs. the calories out we will see the same fat loss.

(So, what we might assume is that protein, carb and fat content are insignificant) Well this is what the study found:

At the same caloric intake, a LOW CARBOHYDRATE diet resulted in significantly GREATER FAT LOSS than a LOW FAT diet.  

 

This is a fraction of the information out there that might get you thinking a little more when you are putting the sweet stuff in your body. And that means carbs. at large.

 

Remember, this is the information that the human body gives us when we put carbohydrates in it. There is a little more to the story, but I did say I would keep it short and sweet. So, if nothing else I keep my word.

MJ

Yuck! Not For Breakfast…

Wednesday, July 9th, 2008

If someone came up to me and asked for my best nutritional tip to start their day, this is what I would say:

“Get a large glass, squeeze ½ fresh lemon in it and fill with WATER. Start drinking. Next, get a large bowl, fill it with MATT’S SLAW (look for attachment), add a handful of raw nuts or seeds on top, drizzle extra virgin olive oil and an organic balsamic vinegar, mix and chow down. This may sound like an “unusual” breakfast, but none of us are usual to begin with. We demand high performance in our work, in our mental acuity, as a” WEEKEND WARRIOR”, playing with our kids…

And this first thing in the morning nourishment meal will help us to do all of this and then some. And here’s why. Water hydrates us. It is ESSENTIAL for optimal health and performance! Lemon and raw vegetables provide all the nutrients i.e. vitamins, minerals, salts, and fiber that each and every cell in our bodies need for High Level Functioning. Secondly, raw vegetable and fresh lemon are foods that create an alkaline (ph above 7) environment in our bodies. Why is this important?

Every cell in our body functions under the complete control of our nervous system. The kicker here is that the nervous system (all of its billions of cells (neurons) thrives in a water medium (bath if you will), that is alkaline (ph above 7). So, a kick butt and happy nervous system means you are kickin’ butt and happy!!!  Cool? I knew this would make sense to you!

 Peace-MJ

SHIFT NUTRITION DAY 30

Monday, July 7th, 2008

SHIFT NUTRITION DAY 30

 

6:45 am-3 oat/blueberry pancakes

              2 caps each of JUICE PLUS orchard and garden blend

 

9:30 am-SUPREME protein bar

 

11:30 am- 2 hard boiled eggs (free range) and 1/3 cantaloupe

 

1:30pm- eggs salad and a serving of organic brown rice

 

3:30pm- handful of organic almonds 1 cup of fresh blueberries

 

6:15pm- handful of assorted nuts and seeds

 

8:00 pm- Steak Fajita

 

2oz. MONAVIE